Вот, кстати, информация по теме, если интересно.
Опять Жуйков на Эхе выступал, сослался на статью Харрисона и др.
Она лежит в свободном доступе(получена 1 февраля, принята 9 февраля, опубликована 6 марта):
John Harrison, Rich Leggett et.al., Polonium-210 as a poison, J. Radiol. Prot. 27 (2007) 17–40
http://www.iop.org/EJ/article/-search=2 ... _1_001.pdf
Abstract
The death of Alexander Litvinenko on 23 November 2006 has brought into
focus scientific judgements concerning the radiotoxicity of polonium-210
(210Po). This paper does not consider the specific radiological circumstances
surrounding the tragic death of Mr Litvinenko; rather, it provides an evaluation
of published human and animal data and models developed for the estimation
of alpha radiation doses from 210Po and the induction of potentially fatal
damage to different organs and tissues. Although uncertainties have not been
addressed comprehensively, the reliability of key assumptions is considered.
Concentrating on the possibility of intake by ingestion, the use of biokinetic and
dosimetric models to estimate organ and tissue doses from 210Po is examined
and model predictions of the time-course of dose delivery are illustrated.
Estimates are made of doses required to cause fatal damage, taking account of
the possible effects of dose protraction and the relative biological effectiveness
(RBE) of alpha particles compared to gamma and x-rays. Comparison of
LD50 values (dose to cause death for 50% of people) for different tissues with
the possible accumulation of dose to these tissues suggests that bone marrow
failure is likely to be an important component of multiple contributory causes
of death occurring within a few weeks of an intake by ingestion. Animal data
on the effects of 210Po provide good confirmatory evidence of intakes and doses
required to cause death within about 3 weeks. The conclusion is reached that
0.1–0.3 GBq or more absorbed to blood of an adult male is likely to be fatal
within 1 month. This corresponds to ingestion of 1–3 GBq or more, assuming
10% absorption to blood. Well-characterised reductions in white cell counts
would be observed. Bone marrow failure is likely to be compounded by damage
caused by higher doses to other organs, including kidneys and liver. Even if the
bone marrow could be rescued, damage to other organs can be expected to prove
fatal.
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